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Phenazepam is a benzodiazepine agonist of the γ-aminobutyric acid A (GABA)-benzodiazepine . Buy research chemicals online”

Description

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Buy Phenazepam powder 30G Online. Phenazepam is a benzodiazepine agonist of the γ-aminobutyric acid A (GABA)-benzodiazepine receptor chloride channel complex. It has been shown to have strong anxiolytic, sedative, anticonvulsive, and hypnotic properties in humans as well as in animal species when administered at 1 mg/kg. At very low doses (10-5 to 10-10 mg/kg), phenazepam has been shown to act as an anxioselective tranquilizer. Buy Phenazepam powder 30G Online.  Order Phenazepam powder 30G Online.

 

Buy Phenazepam powder 30G online, is a benzodiazepine agonist of the γ-aminobutyric acid A (GABAA)-benzodiazepine receptor chloride channel complex. It has been shown to have strong anxiolytic, sedative, anticonvulsive, and hypnotic properties in humans as well as in animal species when administered at 1 mg/kg. At very low doses (10-5 to 10-10 mg/kg), phenazepam has been shown to act as an anxioselective tranquilizer.
Buy Phenazepam for research purposes. Phenazepam produced under German supervision.
trivial name BD 98, Fenazepam, Phenazepam
IUPAC 7-bromo-5-(2-chlorophenyl)-1,3-dihydro-2H-1,4-benzodiazepin-2-one
CAS 51753-57-2
Purity ≥98%
Appearance white powder
Molecular weight 349.6

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Side effects
Side effects include hiccups, dizziness, loss of coordination and drowsiness, along with anterograde amnesia which can be quite pronounced at high doses. As with other benzodiazepines, in case of abrupt discontinuation following prolonged use, severe withdrawal symptoms may occur including restlessness, anxiety, insomnia, seizures, convulsions and death, though because of its long half-life, these withdrawal symptoms may take two or more days to manifest.

Hypertonia is a term sometimes used synonymously with spasticity and rigidity in the literature surrounding damage to the central nervous system, namely upper motor neuron lesions. Impaired ability of damaged motor neurons to regulate descending pathways gives rise to disordered spinal reflexes, increased excitability of muscle spindles, and decreased synaptic inhibition. These consequences result in abnormally increased muscle tone of symptomatic muscles. Some authors suggest that the current definition for spasticity, the velocity-dependent over-activity of the stretch reflex, is not sufficient as it fails to take into account patients exhibiting increased muscle tone in the absence of stretch reflex over-activity. They instead suggest that “reversible hypertonia” is more appropriate and represents a treatable condition that is responsive to various therapy modalities like drug and/or physical therapy.

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